Friday 24 January 2020

Green Peas (Pisum Sativum) Inhibits the Risk of Atherosclerosis induced Heart Diseases

By Kyle J. Norton

Atherosclerosis is a medical condition caused by plaques accumulated on the wall of the arteries, leading to narrowing the arteries in transporting oxygenated blood to the heart and other parts of the body.

Depending on the location of blood obstruction, atherosclerosis can induce different types of heart disease and stroke.

Plaque is the make of cholesterols, calcium, and other toxic substances, including cadmium.

According to the statistics provided by Atherosclerosis Open Acess, 32% of deaths account for atherosclerosis. 13.8% prevalence is among adults of 20 years and above. In 2010, coronary heart disease mortality rate was about 60%.

Atherosclerosis is a slow, progressive disease that may begin as early as childhood, caused by damage to the inner lining of the artery. However, the actual causes of the condition are debatable.

Long term high blood pressure, high cholesterol and triglycerides (a type of fat) in the blood. Particularly, in high levels of insulin in the blood from diabetes and in exposure to chemicals from smoking tobacco.

Patients with early stages of atherosclerosis may not experience any symptoms. However, as the disease progress, patients may have the symptoms of chest pain if the blockage occurs in an artery of the heart (coronary artery) and leg cramps during exercise or walking if the blockage occurs in the legs.

In the advanced stage, if atherosclerosis can lead to a heart attack and stroke, respectively.

Green peas are species of Pisum Sativum belongings to the family Fabaceae and native to western Asia from the Mediterranean Sea to the Himalaya Mountains. The pea is a green, pod-shaped vegetable and a cool-season crop grown in many parts of the world.

On finding a potent ingredient for the treatment of diseases associated with arterial plaques accumulation, researchers investigated the impact of a diet rich in green and yellow vegetables on the development of atherosclerosis.

The study included a mouse model of atherosclerosis, randomized into 2 diet groups: 1) a vegetable-free control diet (n = 53) and 2) the same diet with 30% (w:w) replaced by an equal-parts mixture of freeze-dried peas, green beans, broccoli, corn, and carrots (n = 54), for 16 weeks.


According to the tested analysis,
* Aortic atherosclerosis estimated by cholesteryl ester content, was reduced 38% (P < 0.001) in mice fed the vegetable-rich diet.

* Plasma total cholesterol (-12%), VLDL + ILDL cholesterol (-32%), serum amyloid A (-37%), and body weight (-7%) (all P < 0.01) were also lower in vegetable-rich diet mice at the end of the treatment period. comapred to control.

* In a regression model, a vegetable-rich diet demonstrated antiatherogenic effects of the vegetable diet without explaining the levels of plasma lipoproteins or body weight.

Based on the results, scientists said, " ...a diet rich in green and yellow vegetables inhibits the development of atherosclerosis and may, therefore, lead to a reduction in the risk of coronary heart disease".

Taken altogether, green peas may be considered a functional food for the prevention of atherosclerosis induced heart diseases,  pending to the confirmation of the larger sample size and multicenter human study.


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Author Biography
Kyle J. Norton (Scholar, Master of Nutrition, All right reserved)

Health article writer and researcher; Over 10.000 articles and research papers have been written and published online, including worldwide health, ezine articles, article base, health blogs, self-growth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bioscience, ISSN 0975-6299.

Sources
(1) Green Peas (Pisum Sativum) atherosclerosis by Adams MR1, Golden DL, Chen H, Register TC, Gugger ET. (PubMed)

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