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Monday, 14 November 2016

Antioxidants: Neurodegenrative diseases - Lou Gehrig's diseases: The Effects of Antioxidants on Lou Gehrig's disease

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


Human aging is a biological process, no one can stop, but delay it. It is possible that one person has a physiological younger than his or her biological if one engages in healthy living life style and eating healthily by increasing the intake of good healthy food such as whole grain, fruits, vegetables, beans and legumes, etc. and reducing the consumption of harmful foods, such as saturated fat, trans fat, artificial ingredients, etc.

                      

                 Neurodegenrative diseases

Neurodegeneration is defined as a health conditions of the progressive loss of structure or function of neurons, including death of neurons, includingParkinson’s, Alzheimer’s, and Huntington’s diseases due to genetic mutations, most of which are located in completely unrelated genes.


                                         Lou Gehrig's diseases 

Lou Gehrig's diseases is defined as a condition of neurological disorders that selectively affect the motor neurones caused by the degeneration of neurons located in the two separate anatomical structures of the spinal cord and the cerebral cortex that provide activity of carrying nerve impulses from receptors to the central nervous system.

The Effects of Antioxidants on Lou Gehrig's disease

a. Vitamin B12 (methylcobalamin)
Researcher found that high doses of vitamin B12 (25 mg) as an antioxidant have been shown to improve or slow muscle wasting in the later stages of patients with ALS disease.

b. Vitamin E
Vitamin E beside helps to protect cell membranes from lipid peroxidation damage that reduce the risk of breakdown of the cell membrane, causing ALS.

c. Superoxide dismutase enzyme
Research found that mutations in the superoxide dismutase enzyme can increase the risk ALS in catalyzing the dismutation of superoxide into oxygen and hydrogen peroxide.

d. Cerebral cortex
Researcher found that oxidative protein damage and DNA alternation were found in elevating levels in the cerebral cortex of those with sporadic ALS.

e. Amino acids
study found that diet high in amino acids as antioxidants have shown some promising effect in treating ALS.

f. Etc.