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Saturday, 10 September 2016

Antioxidants: The Effects of Free Radicals on Atherosclerosis of Cardiovascular disease

Kyle J. Norton(Scholar, Master of Nutrients), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.


Human aging is a biological process, no one can stop, but delay it. It is possible that one person has a physiological younger than his or her biological if one engages in healthy living life style and eating healthily by increasing the intake of good healthy food such as whole grain, fruits, vegetables, beans and legumes, etc. and reducing the consumption of harmful foods, such as saturated fat, trans fat, artificial ingredients, etc.

                      

                                 Cardiovascular disease 

Cardiovascular disease or hear diseases are the class of diseases that involve disorder of the heart or blood vessels (arteries and veins),including coronary heart disease (heart attacks), cerebrovascular disease (stroke), raised blood pressure (hypertension), peripheral artery disease, rheumatic heart disease, congenital heart disease and heart failure. Cardiovascular disease kills more than 2,000 Americans everyday and approximately 60 million Americans have heart disease.


                       The Effects of Free Radicals on Atherosclerosis


Atherosclerosis is defined as a condition in which fatty material accumulated along the walls of arteries, leading to thickening or hardening of arterial walls result in blockage eventual of the arteries

Atherosclerosis develops as a result of oxidation of low-density lipoprotein molecules (LDL) or bad cholesterol by free radicals, including reactive oxygen species (ROS). As the oxidated LDL move in the blood stream and comes to contact and damage the arterial wall, the immune immune system by sending specialized white blood cells (macrophages and T-lymphocytes) to absorb the oxidized-LDL forming specialized foam cells. If the white blood cells can not process the oxidized-LDL, they ultimately grow then rupture thus depositing a greater amount of oxidized cholesterol into the artery wall that triggers more white blood cells, continuing the cycle. If the artery becomes inflamed, it causes cholesterol plaque buildup over the affected area, leading to narrowing of the artery that reduces the blood flow and increases blood pressure.
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