Dangerously, In US, Unexpected Poor-Healthy May Lead to Bankruptcy, Researchers Found

By Kyle J. Norton

Recent study suggested that poor health with medical bill piled up and lost of income may contribute to bankruptcy declaration.

The result of the studies were differentiated by numbers of institutes and published on online financial literature, including Massachusetts Institute of Technology.

According to the study medical expenses account for roughly 4 percent of bankruptcy filings among non elderly adults in the U.S., however, bankruptcy filers for medical reason also accompanied with an estimated loss of over 20% of income or 11 percent drop in employment due to hospitalization, particularly, in adults ages 50 and 59.

These results were clearly stated even with a very comprehensive medical insurance without including the the loss of income insurance, people with poor health sometimes during their life time may have a substantial risk of bankruptcy.

In the joint study conducted by numbers of respectable scientists, using the data from the LIVESTRONG 2012 survey of 4,719 cancer survivors ages 18-64, in investigated the proportions of survivors who reported going into debt or filing for bankruptcy as a result of cancer, as well as the amount of debt incurred indicated that

* Over 30% of survivor had gone into debt with 55 percent incurred obligations of $10,000 or more.

* 3 percent had filed for bankruptcy in compared to 4% in common statistics.

Further analysis, also discovered, cancer survivors who were younger, had lower incomes, and had public health insurance were more likely to go into debt or file for bankruptcy in compared to those who were older, had higher incomes, and had private insurance.

These results one more time indicated the need to have a comprehensive medical insurance and a long time income replacement policy. 

Truly, researchers should also need to take into account of younger adult who can not afford or was not approved of disability insurance due to job related activities.

The differentiation of the above were supported by evaluation conducted by the numbers of instututes professors, including Massachusetts General Hospital, Boston; Danielle Blanch-Hartigan, Bentley University, Waltham.

In a total of 1,202 adult cancer survivors diagnosed or treated at ≥ 18 years of age from the 2011 Medical Expenditure Panel Survey Experiences With Cancer questionnaire. Material financial hardship was measured by ever (1) borrowing money or going into debt, (2) filing for bankruptcy, (3) being unable to cover one's share of medical care costs, or (4) making other financial sacrifices because of cancer, its treatment, and lasting effects of treatment, researchers found that material financial and psychological hardship was more common in cancer survivors age 18 to 64 years in compared to those ≥ 65 years of age.

Patients of 18 to 64 years must deal with more stressful events, including family to support, mortgage to pay, employment, loss of income in compared to retired population without income to worry.

Moreover, cancer survivors age 18 to 64 years who were younger, female, nonwhite, and treated more recently and who had changed employment because of cancer were significantly more likely to report any material financial hardship.

This result can be easily to interpret as these group of people are most likely to work in the low paying jobs with no insurances, implying cancer survivors who were uninsured, had lower family income, and treated more recently were more likely to report psychological financial hardship.

Taken together, people who are not insured by a comprehensive package of medical and disability insurance are likely to face psychological and financial hardship and risk of bankruptcy,

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Sources
(1) How often do medical problems lead to bankruptcy? by Massachusetts Institute of Technology
(2) For Working-Age Cancer Survivors, Medical Debt And Bankruptcy Create Financial Hardships by Banegas MP1, Guy GP Jr2, de Moor JS3, Ekwueme DU4, Virgo KS5, Kent EE6, Nutt S7, Zheng Z8, Rechis R9, Yabroff KR(PubMed)
(3) Financial Hardship Associated With Cancer in the United States: Findings From a Population-Based Sample of Adult Cancer Survivors by Yabroff KR1, Dowling EC2, Guy GP Jr2, Banegas MP2, Davidoff A2, Han X2, Virgo KS2, McNeel TS2, Chawla N2, Blanch-Hartigan D2, Kent EE2, Li C2, Rodriguez JL2, de Moor JS2, Zheng Z2, Jemal A2, Ekwueme DU2(PubMed)

Add a Portion of Lentil Optimize Your Immunity Against Pathogenic Infection

By Kyle J. Norton

Lentil may have a profound and positive effect in modulated immune response in acute phase of infection, some scientist suggested

Lentil is a type of pulse and tiny legumes that can be cooked quickly and rich in protein and fiber, belongings genus Lens the Fabaceae family, native to Middle East.

Immune response is a natural reaction of immune system to protect our body against invasion of foreign substances.

In acute phase of infection, immune system initiated production of of proinflammatory Th1 cytokines in activated interleukin-12 (IL-12) and gamma interferon (IFN-γ) and other pro inflammatory factors such as TNF beta in response to detect, engulf and destroy pathogens and apoptotic cells through macrophages.

These activities played an important in induced apoptosis and reduced proliferation of bacteria or virus by attaching to the surface of the injure cells to initiation of alternation. however, over production of pro inflammatory expression may indeed cause substantial adverse effects in tissue destruction, and, in some cases, shock and death.

Type 1 T helper (Th1) in response to tissue damage and injure also activated macrophages which are responsible for cell-mediated immunity and phagocyte-dependent protective activity.


In the concerns of lectins adverse effecs may pose a potential threat to consumers due to their capacity to induce histamine release from basophils, after investigating over 16 common dietary produceds containing lectins researchers found that most plant lectins induce human basophils to produce interleukin 4 (IL-4) a anti inflammatory cytokine, which plays an important role in stimulated secretion of T cells and IL-13 another cytokines works conjunction with IL-4 in promoted the expression of T helper 2 (Th2) responses and regulation of Immunoglobulin E (IgE) synthesis.


All these expressed a significant effect of dietary lectin in reduced over expression of pro inflammatory factors in immune system during acute phase of infection in induced further cell damage and death in the infectious site through secretion of anti inflammatory cytokines without decreasing the immune protective activities.


Truly, further examine also found that concanavalin A, lentil lectin, phytohemagglutinin, , triggered basophils to release IL-4 at concentrations of up to 1 ng/10(6) basophils which in turn orchestrated the release of IL-13 and histamine and levels of IL-4 and IL-13 release reached a maximum after 4-6 h and more than 18 h, after injection, , respectively.


These activities demonstrated a significant effect of lentil lectin in inducing early IL-4 required to switch the immuneresponse towards a Th2 response and type I allergy, involving immunoglobulin E (IgE)–mediated release of histamine and other mediators from mast cells and basophils.

Indeed, the relative balance between Th1 and Th2 cytokines appears crucial, as Th1 cells activated immune response during the early state of body injure or damage through evoking cell-mediated immunity and phagocyte-dependent inflammation, in stimulated production of several pro inflammatory cytokines such as interferon (IFN)-gamma, interleukin (IL)-2 and tumor necrosis factor (TNF)-beta which may also cause damage to the cell of the infectious region, leading to permanently additional damage to the tissues and Th2, under response to over expression of pro inflammatory cytokines stimulated the production of IL-4, IL-5, IL-6, IL-9, IL-10, and IL-13 of anti inimflammatory mytokines in evoked strong antibody responses (including those of the IgE class) and eosinophil accumulation, and inhibited several functions of phagocytic cells as indicated above.


Taken together, Lentil lectin may have a anti inflammatory effect in reduced over expression of pro inflammatory factors in initiation of symptoms of fever, headache, swelling, redness, loss of appetite, muscle stiffness through activating production of anti inflammatory mytokines.


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Holistic System In Existence That Will Show You How To
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FOOD HACK for Weight Loss
A Simple Cooking Technique That Cuts The Calories & Glycemic
Impact In Rice, Pasta, And Potatoes In Half

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Author Biography
Kyle J. Norton
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.

Sources
(1) Role of Th1 and Th2 Cytokines in Immune Response to Uncomplicated Plasmodium falciparum Malaria by Donato Torre,1,* Filippo Speranza,1 Massimo Giola,1 Alberto Matteelli,2 Roberto Tambini,1 andGilberto Biondi1(PMC)
(2) Dietary lectins can induce in vitro release of IL-4 and IL-13 from human basophils by Haas H1, Falcone FH, Schramm G, Haisch K, Gibbs BF, Klaucke J, Pöppelmann M, Becker WM, Gabius HJ, Schlaak M.(PubMed)
(3) T-cell subsets (Th1 versus Th2) by Romagnani S1(PubMed)

The Story of Vitamin C in Expression of Breast Cancer

By Kyle J. Norton


Vitamin C, also known as L-ascorbic acid, is a water-soluble vitamin, found in fresh fruits, berries and green vegetables. It is best known for its free radical scavengers activity and regenerating oxidized vitamin E for immune support.

Epidemiological studies linking vitamin C in reduced risk of breast cancer may be inconclusive(1)(1a)(1b), but no doubt in acceptance of improved quality of life(2).

Macro nutrients intake may form an important parts in breast cancer patients in providing vital support for treatment.(3). There was a report of intake of supplementation of multiple vitamin, beta-carotene, vitamin C, vitamin E and zinc in postmenopausal women for 10 or more years may protect women from developing breast cancer(3a). 

Women with breast cancer in the Indian population, were found to have a lower levels of mean vitamin C, vitamin E and selenium than controls. if the levels of mean vitamin C, vitamin E and selenium increased by 1 unit, the risk of breast cancer was reduced by 7%(3b).

In breast cancer survival, dietary vitamin C intake before breast cancerdiagnosis may be associated with breast cancer survival. but not in post-diagnosis(4). 

High intake of ascorbic acid was in associated to reduce risk of breast cancer incidence in overweight women and women with high consumption of linoleic acid (average consumption of more than 6 grams of linoleic acid per day)(5) and insignificant risk in other breast cancer patients(6). 

On inflammation in cancer patients, high dose intravenous ascorbic acid therapy, decreased the levels of C-reactive protein thus reduced inflammation correlated with decreases in tumor marker levels(7). 

Vitamin C supplements and Anthocyanin (Ixor®) at a dose of 2 tablets/day, starting from 10 days before the radiation treatment until 10 days after the end of treatment was found to be protective against skin damage to patient undergoing adjuvant chemotherapy(8).

In estrogen-induced breast carcinogenesis, vitamin C (Vit C) and butylated hydroxyanisole (BHA) found to be effective in inhibition of 17β-estradiol (E2)-mediated oxidative stress and oxidative DNA damage by preventing the decreasing NRF2(antioxidant response pathway) and OGG1(base excision repair.) levels(9). 

In the study of the same but in MCF-10A cells, the combination also decreased E2-mediated increase in 8-OHdG(Marker detected in cancer patients) levels in the mammary tissues, induced SOD3 (Extracellular superoxide dismutase [Cu-Zn]) through NRF2 Pathway to defense against oxidative stress and in the prevention of estrogen-mediated breast cancer(10).

An increased expression of the miR-93(Regulate Expression of Tumor Suppressor Gene) was found in 17β-estradiol (E2)-treated mammary tissues and in human breast cell lines, treatment with vitamin C reverted E2-mediated increase in miR-93 levels by upregulating expression NRF2 antioxidant response pathway(11). 

In 4T1 breast cancer cells in vitamin C-deficient mice, Ascorbic acid delayed the progress of metastasis, tumor growth and inflammatory cytokine secretion (decreased serum inflammatory cytokine interleukin (IL)-6) as well as enhanced encapsulation of tumors(12).

 In L-ascorbate (L-ascorbic acid, vitamin C), increasing the concentration exhibited the autophagic damage to functional SVCT-2(antibody) sensitizes breast cancer cells(13). 

In B16F10, L-ascorbate also caused induction of a prooxidant state, subsequent reduction in mitochondrial membrane potential to induced apoptosis in a caspase-8(Cell apoptosis)-independent manner(14).

 In the usage of glucan, resveratrol and vitamin C, the combination showed the strongest activator of phagocytosis (immune cell activation) and antibody formation to suppress the growth of breast and lung tumors, through stimulation of apoptosis(15). 

In 4T1 cancer cell line, combined with ascorbate, Mn(III)N-alkylpyridylporphyrins (MnPs) inhibited cancer cells via peroxide produced outside of the cell through enhancing tumour oxidative stress and tumor growth suppression(16). 

In Ataxia telangiectasia mutated (ATM) diplotype on the breast cancer, vitamin C enhanced the increase of ATM to reduce the risk of breast cancer.(17). 

In E(2) metabolism and oxidant stress in involved in estrogen-induced breast cancer development, vitamin C reducesd the incidence of estrogen-induced mammary tumors, increased tumor latency and decreases oxidative stress in vivo(18). 

In SK-BR3 and Hs578T breast cancer cell lines, Vitamin C treatment induced AIF(apoptosis-inducing factor) mediation of cell death pathway of the breast cancer cell lines independent to caspase pathway(19).

In human breast cancer cell line MCF-7, combination of Retinoic acid and ascorbic acid inhibited the proliferation of human breast cancercells through altering their gene expression related to antioxidation processes and the proliferation inhibitory pathway(20).

Taking all together, without going into reviews, vitamin C is effective in reduced risk and  treatment of breast cancer. 

Daily ingestion of high-dose vitamin C may be considered safe, but in rare incidence, overdoses in a prolonged period of time, may cause intra-renal oxalate crystal deposition, a fatal nephrotoxicity(21)(22).


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References
(1) Vitamin C suppresses cell death in MCF-7 human breast cancer cells induced by tamoxifen by Subramani T, Yeap SK, Ho WY, Ho CL, Omar AR, Aziz SA, Rahman NM, Alitheen NB.(PubMed)
(1a) Vitamin supplement consumption and breast cancer risk: a review by Misotti AM, Gnagnarella P.(PubMed)
(1b) Dietary fiber, vitamins A, C, and E, and risk of breast cancer: a cohort study by Rohan TE, Howe GR, Friedenreich CM, Jain M, Miller AB.(PubMed)
(2) Intravenous vitamin C administration improves quality of life in breast cancer patients during chemo-/radiotherapy and aftercare: results of a retrospective, multicentre, epidemiological cohort study in Germany by Vollbracht C, Schneider B, Leendert V, Weiss G, Auerbach L, Beuth J.(PubMed)
(3) Nutritional assessment of selected patients with cancer.

Surwillo A, Wawrzyniak A.(PubMed)
(3a) Antioxidants and breast cancer risk- a population-based case-control study in Canada by Pan SY, Zhou J, Gibbons L, Morrison H, Wen SW; Canadian Cancer Registries Epidemiology Research Group [CCRERG].(PubMed)
(3b) Association between breast cancer and vitamin C, vitamin E and selenium levels: results of a case-control study in India by Singh P, Kapil U, Shukla NK, Deo S, Dwivedi SN.(PubMed)
(4) Vitamin C intake and breast cancer mortality in a cohort of Swedish women by Harris HR, Bergkvist L, Wolk A.(PubMed)
(5) Dietary antioxidant vitamins, retinol, and breast cancer incidence in a cohort of Swedish women by Michels KB, Holmberg L, Bergkvist L, Ljung H, Bruce A, Wolk A.(PubMed)
(6) Vitamins C and E, retinol, beta-carotene and dietary fibre in relation to breast cancer risk: a prospective cohort study. by Verhoeven DT, Assen N, Goldbohm RA, Dorant E, van 't Veer P, Sturmans F, Hermus RJ, van den Brandt PA.(PubMed).
(7) Effect of high-dose intravenous vitamin C on inflammation in cancerpatients by Mikirova N, Casciari J, Rogers A, Taylor P.(PubMed)
(8) Skin toxicity from external beam radiation therapy in breast cancerpatients: protective effects of Resveratrol, Lycopene, Vitamin C and anthocianin (Ixor®) by Di Franco R, Calvanese M, Murino P, Manzo R, Guida C, Di Gennaro D, Anania C, Ravo V.(PubMed)
(9) Antioxidant-mediated up-regulation of OGG1 via NRF2 induction is associated with inhibition of oxidative DNA damage in estrogen-induced breast cancer by Singh B, Chatterjee A, Ronghe AM, Bhat NK, Bhat HK(PubMed).
(10) Superoxide dismutase 3 is induced by antioxidants, inhibits oxidative DNA damage and is associated with inhibition of estrogen-induced breast cancer by Singh B, Bhat HK.(PubMed)
(11) MicroRNA-93 regulates NRF2 expression and is associated with breast carcinogenesis by Singh B, Ronghe AM, Chatterjee A, Bhat NK, Bhat HK.(PubMed)
(12) Ascorbate supplementation inhibits growth and metastasis of B16FO melanoma and 4T1 breast cancer cells in vitamin C-deficient mice by Cha J, Roomi MW, Ivanov V, Kalinovsky T, Niedzwiecki A, Rath M.(PubMed)
(13) SVCT-2 in breast cancer acts as an indicator for L-ascorbate treatment by Hong SW, Lee SH, Moon JH, Hwang JJ, Kim DE, Ko E, Kim HS, Cho IJ, Kang JS, Kim DJ, Kim JE, Shin JS, Jung DJ, Jeong YJ, Cho BJ, Kim TW, Lee JS, Kang JS, Hwang YI, Noh DY, Jin DH, Lee WJ.(PubMed)
(14) L-ascorbic acid (vitamin C) induces the apoptosis of B16 murine melanoma cells via a caspase-8-independent pathway by Kang JS, Cho D, Kim YI, Hahm E, Yang Y, Kim D, Hur D, Park H, Bang S, Hwang YI, Lee WJ.(PubMed)
(15) Combination of glucan, resveratrol and vitamin C demonstrates strong anti-tumor potential.

Vetvicka V, Vetvickova J.(PubMed)
(16) Cytotoxic effects of Mn(III) N-alkylpyridylporphyrins in the presence of cellular reductant, ascorbate by Ye X, Fels D, Tovmasyan A, Aird KM, Dedeugd C, Allensworth JL, Kos I, Park W, Spasojevic I, Devi GR, Dewhirst MW, Leong KW, Batinic-Haberle I.(PubMed)
(17) Antioxidant vitamins intake, ataxia telangiectasia mutated (ATM) genetic polymorphisms, and breast cancer risk by Lee SA, Lee KM, Lee SJ, Yoo KY, Park SK, Noh DY, Ahn SH, Kang D.(PubMed)
(18) Vitamin C and alpha-naphthoflavone prevent estrogen-induced mammary tumors and decrease oxidative stress in female ACI rats by Mense SM, Singh B, Remotti F, Liu X, Bhat HK.(PubMed)
(19) Ascorbate (vitamin C) induces cell death through the apoptosis-inducing factor in human breast cancer cells by Hong SW, Jin DH, Hahm ES, Yim SH, Lim JS, Kim KI, Yang Y, Lee SS, Kang JS, Lee WJ, Lee WK, Lee MS.(PubMed)
(20) Retinoic acid and ascorbic acid act synergistically in inhibiting human breast cancer cell proliferation by Kim KN, Pie JE, Park JH, Park YH, Kim HW, Kim MK.(PubMed)
(21) Fatal vitamin C-associated acute renal failure by McHugh GJ, Graber ML, Freebairn RC.(PubMed)
(22) Ascorbic acid overdosing: a risk factor for calcium oxalate nephrolithiasis by Urivetzky M, Kessaris D, Smith AD.(PubMed)

Warning to: Fish Omega 3 Supplement Consumers, Beware of 14 Side Effects

Kyle J. Norton

Omega-3 fatty acids are phytochemincals in the class of lipids, found abundantly in deep sea fish, dark-green leafy vegetables, grains, legumes, nuts,.....

Fish oil omega-3 fatty acids, contained mainly eicosapentaenoic acid and docosahexaenoic acid, although the supplement has been found to process some significant effects in the management and treatment of several diseases, many of which are inflammatory in nature. However, the supplement may induce some outlined adverse effects.

1. Altered platelet function Omage 3 fatty acid made from fish may have a strong implication on plaletet function due to the presence of eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) in increased occlusion time and sharply reduced accumulation of fibrin under flow conditions(1)(2).

Therefore, people with normal bleeding problem should be avoid taking the supplement(2).

2. Gastrointestinal adverse effects

Although Omega-3 fatty acids may have a potential in triglycerides lowering, reduction of inflammatory indices, immunomodulation, anti- thrombotic effects, particularly in gastro-protective activity. However, when the healthy supplement taken together with cortisone may cause gastrointestinal bleeding in induction of duodenum ulcer.

This result may be attributed by cortisone in increased oxidation in render them proinflammatory(3).

Other researchers suggested that over doses of omega-3 fatty acids may enhance intestinal production and secretion of intestinal alkaline phosphatase (IAP), in induction of the induces changes of gut bacteria composition resulting in decreased lipopolysaccharide production and gut permeability(7).

3. Detrimental Effects on Wound Healing

In the observation of epidermal growth factor receptor (EGFR)-mediated signaling is required for optimal intestinal wound healing, researchers found that during the early response to intestinal wounding, DHA and EPA uniquely delay the activation of key wound-healing processes in the colon through at least in part suppression of EGFR function(4).

4. Increased risk of lipid peroxidation

Omega 3 fatty acide may interact with reactive oxygen species ROS in inscreased risk of lipid peroxidation in induction of ethane expression in human breath(5).

5. Toxin exposure

Omega 3 fatty acid produced by large amounts of fish may result in adverse effect due to the potential presence of environmental toxins such as mercury, polychlorinated biphenyls, dioxins,.... (8).

Moreover, omega-3 fatty acid preparation may undergo oxidation incontribution to patient intolerance and potential toxicity(8).

6, Weight gain

Intake of omega 3 fatty during pregancy may have a potential effect in induced weight gain of male offspring a poor outcomes associated with high maternal weight gain early in pregnancy(10)..

7. Altered immune function

Omega 3 fatty acide with abubdant arachidonic acid, EPA and DHA contents may alter the balanced pattern of production of eicosanoids and probably also of docosanoids and resolvins, affecting phagocytosis, T-cell signaling and antigen presentation capability.(11).

8. Glycemic control
According to a randomized double-blind crossover study in compared 6 wk of n-3 fatty acid supplementation (12 g fish oil) and corn plus olive oil, long term intake of 3 fatty acid supplementation expressed in increased glycerol gluconeogenesis in contribution to deterioration of glycemic control(12).

However, during the 6 weeks of experiment, researchers also found intake of n-3 fatty acid supplementation lowers triacylglycerols in patients with non-insulin-dependent diabetes mellitus without worsening glycemic control(12).

Drug interactions
Intake of Omega-3 fatty acids
9. May increase the effects of blood thinning medications, including aspirin, warfarin (Coumadin), and clopedigrel due to its anti coagulative activity(13).

10. May increase fasting blood sugar levels, thus reducing the effect of blood sugar lowering medications, such as ,glipizide (Glucotrol and Glucotrol XL), glyburide (Micronase or Diabeta), glucophage (Metformin)(13).

11. Lower the effect of Cholesterol-lowering medications due to the alternation of ratio of good and bad cholesterol(13).

12. Pregnancy and 13. Lactation

Consumption of the nutrients omega-3 fatty acids (omega-3 FA) during pregnancy and lactation is considered beneficial to fetal and infant development. However, according to the animal study by the Wayne State University School of Medicine, Consuming or administering large or inadequate amounts of omega-3 FA during pregnancy and lactation may induce the potential for adverse effects on infant development(6).

14. Impair host resistance to intracellular pathogens

In the study of Wild-type and fat-1 transgenic mice infected with virulent H37Rv M. tuberculosis via the aerosol route, observartion of bacteriological and histopathological changes in lungs, researchers suggested that endogenous production of n-3 PUFAs in fat-1 mice increases their susceptibility to tuberculosis due to impaired bacterial control in cells and reduced proinflammatory cytokine secretion, impaired oxidative metabolism, and diminished M. tuberculosis-lysotracker colocalization within phagosomes(9).

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Arthritis Is Curable
You Can Eliminate Osteoarthritis
By addressing the Underlying Causes through Clinical Trials and Studies

Ovarian Cysts And PCOS Elimination
Holistic System In Existence That Will Show You How To
Permanently Eliminate All Types of Ovarian Cysts Within 2 Months

FOOD HACK for Weight Loss
A Simple Cooking Technique That Cuts The Calories & Glycemic
Impact In Rice, Pasta, And Potatoes In Half

Back to Kyle J. Norton Home page http://kylejnorton.blogspot.ca

Author Biography
Kyle J. Norton (Scholar, Master of Nutrition, All right reserved)
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.

Sources

(1) Potential adverse effects of omega-3 Fatty acids in dogs and cats by Lenox CE1, Bauer JE.(PubMed)

(2) Exogenous modification of platelet membranes with the omega-3 fatty acids EPA and DHA reduces platelet procoagulant activity and thrombus formation byLarson MK1, Tormoen GW, Weaver LJ, Luepke KJ, Patel IA, Hjelmen CE, Ensz NM, McComas LS, McCarty OJ.(PubMed)

(3) Gastrointestinal bleeding after high intake of omega-3 fatty acids, cortisone and antibiotic therapy: a case study by Detopoulou P1, Papamikos V.(PubMed)

(4) Inhibitory effects of omega-3 fatty acids on injury-induced epidermal growth factor receptor transactivation contribute to delayed wound healing by Turk HF1, Monk JM, Fan YY, Callaway ES, Weeks B, Chapkin RS(PubMed)

(5) Increased levels of ethane, a non-invasive marker of n-3 fatty acid oxidation, in breath of children with attention deficit hyperactivity disorder by Ross BM1, McKenzie I, Glen I, Bennett CP.(PubMed)

(6) Reduced auditory acuity in rat pups from excess and deficient omega-3 fatty acidconsumption by the mother by Church MW1, Jen KL, Stafferton T, Hotra JW, Adams BR.(PubMed)

(7) A host-microbiome interaction mediates the opposing effects of omega-6 and omega-3 fatty acids on metabolic endotoxemia by Kaliannan K1, Wang B1, Li XY1, Kim KJ1, Kang JX1.(PubMed)

(8) Safety considerations with omega-3 fatty acid therapy by Bays HE(PubMed)

(9) Transgenic mice enriched in omega-3 fatty acids are more susceptible to pulmonary tuberculosis: impaired resistance to tuberculosis in fat-1 mice by Bonilla DL1, Fan YY, Chapkin RS, McMurray DN.(PubMed)

(10) Maternal Weight Gain Regulates Omega-3 Fatty Acids in Male, Not Female, Neonates by O'Tierney-Ginn PF1, Gillingham M2, Fowler J3, Brass E4, Marshall NE5,6, Thornburg KL(PubMed)

(11) Immunomodulation by omega-3 fatty acids by Calder PC(PubMed).
(12) Dietary supplementation with n-3 fatty acids increases gluconeogenesis from glycerol but not hepatic glucose production in patients with non-insulin-dependent diabetes mellitus by Puhakainen I1, Ahola I, Yki-Järvinen H.(PubMed)
(13) Possible Interactions with: Omega-3 Fatty Acids by Pennstate University

Food Therapy: Intake Fennel to Optimize the Digestive Functioning in absorbed Nutrients and Fluids

Kyle J. Norton

Fennel may have a therapeutic and enhanced effect in improve digestive system in absorbed nutrients and fluids from foods intake, some scientists suggested.

The result of this efficacy was published online by several respectable institutes, including the University of Illinois at Chicago and Afyon Kocatepe University.

Fennel (Foeniculum vulgare) is a plant species of genus, belongings to Apiaceae (Umbelliferae), native to the Mediterranean, used in traditional and herbal medicine as warming, carminative, antispasmodic, antidepressant agent and to stimulate the appetite, ease indigestion, soothe coughing, reduce intestinal spasms, to regulate the menstrual cycle and relieve PMS,...

Nutrients
1. Carbohydrates
2. Fiber
3. Protein
4. Thiamine (Vitamin B1)
5. Riboflavin (Vitamin B2)
6. Niacin (Vitamin B3)
7. Pantothenic acid (B5)
8. Vitamin B6
9. Folate (Vitamin B9)
10. Vitamin C
11. Calcium
12. Copper
13. Iron
14. Magnesium
15. Molybdenum
16. Phosphorus
17. Potassium
18. Zinc
19. Etc.


According to the study, researchers set out a criteria and guideline to investigate the effect of fennel in gram-negative bacterium Helicobacter pylori (HP), the microorganisms has been associated with the development of gastritis and peptic ulcer disease to confirm the theory of used in traditional medicine fennel to treat a wide range of ailments, including gastrointestinal (GI) disorders such as dyspepsia, gastritis and peptic ulcer disease (PUD), application of methanol extracts of botanicals with a MIC of 50 microg/mL included Achillea millefolium, Foeniculum vulgare (seed), exerted a significantly inhibitory effect against all susceptibility of 15 HP strains.

These results suggested that injection of fennel displayed a potential in balanced the ratio of micro organisms in the gastrointestinal tract, thus improve the function in absorbed and distributed nutrients to the body and reassured the herbal medicine used in practice for treatment of numbers symptoms of 

digestive disorder.

Furthermore, in animal model with rat induced gastric lesions by injection of ethanol, application of aqueous extracts of Foeniculum vulgare (FVE) showed a strong activity in inhibited the ulcerogenic expression through its antioxidant activities..

In observation of the efficacy of 60 minutes pre administration of the doses of 75, 150 and 300 mg/kg, with famotidine, histamine H₂ receptor antagonist with function to inhibit stomach acid production, used at the dose of 20 mg/kg, before injection of ethanol, researchers found that

* Pre treatment of all dose exerted a significant gastric protective effect in reduced injure and damage caused by injection of ethanol.

* Fennel extract demonstrated a theurateutic activity linearly in compared to other doses and control at dose of 300 mg/kg 

* These activities was associated to reduce the malondialdehyde (MDA) levels, a marker in expression of severe levels of oxiadive stress.

Additional analysis, also suggested that fennel stimulated gastro-protect effect through its antioxidants and facilitated production of antioxidants found in the body tissues in inhibited the over expression of free radicals, Thus maintaining the ratio of micro organisms in the digestive tract and its functioning.

After, taking into account of other co and con founders, researchers also suggested that the result in part may also attributed to antioxidant in ameliorated lipid peroxidation.

The above differentiation were supported by the joint report lead by the Joint Keio University in the investigation of fennel properties in associated to functional dyspepsia (FD), a functional gastrointestinal disorder characterized by persistent or recurrent postprandial upper abdominal discomfort and epigastric pain.

Truly, Delayed gastric emptying and impaired gastric accommodation was found to play important roles in the development of FD. 

Anethole (1-methoxy-4-((E)-propenyl)-benzene), a major component of fennel essential oil, in in rodents exerted a huge effect in improved clonidine-induced delayed gastric emptying without harming to normal gastric emptying in mice, orally.

Also, application of fennel oil plus Anchu-san (a Japanese herbal medicine containing anethole) also demonstrated a important activity in reduced risk of functional dyspepsia (FD) through restoring the delayed gastric emptying.

In other words,  anethole processes a gastro protective efficacy through stimulating gastric accommodation. 

Taken together, whole fennel, fennel essential oil and its bioactive compound anethole may be used as secondary application in reduced risk and treatment of digestive disorder. Adding a portion of fennel in your diet may improve the digestive function in absorption nutrients and fluids from the food intake.

However, intake of supplement should be taken with extreme care as overdose may cause acute liver toxicity.

For More information of yoga lessons tailor to a complete well being for women, please visit: YOGA FOR WOMEN

Arthritis Is Curable

You Can Eliminate Osteoarthritis
By addressing the Underlying Causes through Clinical Trials and Studies

Ovarian Cysts And PCOS Elimination
Holistic System In Existence That Will Show You How To
Permanently Eliminate All Types of Ovarian Cysts Within 2 Months

FOOD HACK for Weight Loss
A Simple Cooking Technique That Cuts The Calories & Glycemic 
Impact In Rice, Pasta, And Potatoes In Half  

Back to Kyle J. Norton Home page http://kylejnorton.blogspot.ca

Author Biography
Kyle J. Norton (Scholar, Master of Nutrients, All right reserved)
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.

Sources
(1) In vitro susceptibility of Helicobacter pylori to botanical extracts used traditionally for the treatment of gastrointestinal disorders by Mahady GB1, Pendland SL, Stoia A, Hamill FA, Fabricant D, Dietz BM, Chadwick LR.(PubMed)
(2) Beneficial effects of Foeniculum vulgare on ethanol-induced acute gastric mucosal injury in rats by Birdane FM1, Cemek M, Birdane YO, Gülçin I, Büyükokuroğlu ME.(PubMed)
(3) Anethole restores delayed gastric emptying and impaired gastric accommodation in rodents by Asano T1, Aida S2, Suemasu S2, Mizushima T3.(PubMed)

6 Herbal Remedies to Detox and Nourish Your Kidneys

American diet contains high saturated fat, trans fat, and other harmful substances such as sugar, caffeine, pesticides, additive substances, and less in fruits and vegetables.

These types of harmful substances is toxic.  Our body’s detoxifying functions try to get rid of them everyday.

When they fail it causes a disruption of hormone production, damaging our body’s detoxification organs and weakens our immune system resulting in hormone imbalance and many chronic illness consequently.

The below are outlined of 6 herbs with therapeutic effects to detox and nourish your kidneys

1. Dandelion leaf
Dandelion is a rich source of vitamins A, B complex, C, and D, as well as minerals such as iron, potassium, and zinc.

Dandelion root has been used for centuries in traditional Chinese medicine in treating liver disease by stimulating the removal of liver toxins. Dandelion leaf on the other hand are used as a diuretic to stimulate the excretion of urine in removing the toxin accumulated in the kidney.

2. Echinacea
Commonly called purple coneflower.

The herb contains a wide variety of chemicals that not only helps to strengthen the immune system but also stimulates the removal of toxins that causes kidney inflammation and infection.

3. Corn silk
Corn silks are the silky fibers that are found under the leaves and on top of an ear of corn. It contains flavonoids, essential oil and minerals that helps effectively in treating acute and chronic inflammation of the urinary system caused by toxins accumulated in the kidney.

4. Artichoke
Artichoke is a perennial thistle originating from southern Europe containing thiamin, riboflavin, niacin.

Antioxidants found in Artichoke not only strengthen our immune system, they also treat kidney diseases by reducing protein expression in the urine caused by toxins through poor diet for a prolonged period of time.

5. Golden rod
Golden rod is a yellow flowering plant in the family of Asteraceae.

Golden rod has been used in traditional medicines as a kidney tonic, and treating urinary infections and stones. Golden rod juice also helps to cleanse of kidney and bladder caused by toxins and inflammation.

Arthritis Is Curable
You Can Eliminate Osteoarthritis
By addressing the Underlying Causes through Clinical Trials and Studies

Ovarian Cysts And PCOS Elimination
Holistic System In Existence That Will Show You How To
Permanently Eliminate All Types of Ovarian Cysts Within 2 Months

FOOD HACK for Weight Loss
A Simple Cooking Technique That Cuts The Calories & Glycemic
Impact In Rice, Pasta, And Potatoes In Half

Back to Kyle J. Norton Home page http://kylejnorton.blogspot.ca

How to Improve Your Brain Functioning Effectively

By Kyle J. Norton

Scientists may have found a food whole plant in exerting neuroprotective effect, the renowned institute study suggested.

According to the MVP Samaj’s College of Pharmacy, methinic extract of beet fed to experiment animal showed a significant effect in increased motor function at 200, and 300 mg/kg, in doses depending manner.

Neuroprotection is an action used to ensure the brain normal function against nerve cells damage, degeneration in inducing impairment.

Beet, best known as the beetroot or garden beet and belonging to the amaranth family, is a perennial plant with leafy stems growing to 1–2 m tall.

According to the MVP Samaj’s College of Pharmacy, methinic extract of beet fed to experiment animal showed a significant effect in increased motor function at 200, and 300 mg/kg, in doses depending manner.


Oral administrated animal also showed a strong improvement of intensity of muscular rigidity, duration of catalepsy, akinesia, the number of tremors, locomotor activity and grip strength.


The efficacy of the extracted in contribution of brain health may be a result of present phytochemicals in restoring the defensive antioxidant enzymes in the brain against level of lipid peroxidation (LPO), a leading cause of neuro degeneration.

More interestingly, Betalains, major phytochemicals isolated from beetroot also expressed a extraordinary neuro protective effect in preventing oxidative processes in contribution to the early onset of several degenerative diseases.

In the study of betanin bioavailability in four volunteers with consumption 300 mL of red beet juice, containing 120 mg of the antioxidant, reseraches found that the phytochemical showed a protection against certain oxidative stress-related disorders in humans after absorbed from the gut and identified in urine after 2-4 h, particularly in inhibited lipid peroxidation, a leading cause of neuro degenerative disease.

Dr. Kanner J, the lead author said, “Antioxidant nutrients from fruits and vegetables are believed to be a class of compounds that exert their effects in humans by preventing oxidative processes which contribute to the onset of several degenerative diseases”.



Collectively, Whole food beetroot or beet juice may be used as therapeutic agent for preventing the early onset and treatment of neuro degenerative disease through protection of neuro cells against oxidation.

Arthritis Is Curable
You Can Eliminate Osteoarthritis
By addressing the Underlying Causes through Clinical Trials and Studies

Ovarian Cysts And PCOS Elimination
Holistic System In Existence That Will Show You How To
Permanently Eliminate All Types of Ovarian Cysts Within 2 Months

FOOD HACK for Weight Loss
A Simple Cooking Technique That Cuts The Calories & Glycemic
Impact In Rice, Pasta, And Potatoes In Half

Author biography
Kyle J. Norton, Master of Nutrients
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it's news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.

Kyle J. Norton(Scholar, Master of Nutrition), all right reserved.
Health article writer and researcher; Over 10.000 articles and research papers have been written and published on line, including world wide health, ezine articles, article base, healthblogs, selfgrowth, best before it’s news, the karate GB daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada – Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as international journal Pharma and Bio science, ISSN 0975-6299.

Sources
(1) Neuroprotective potential of Beta vulgaris L. in Parkinson’s disease by Nade VS1, Kawale LA1, Zambre SS1, Kapure AB1.(PubMed)
(2) Betalains–a new class of dietary cationized antioxidants by Kanner J1, Harel S, Granit R.(PubMed)