Epidemiological studies, linking coffee consumption daily and regularly associated with risk of gastritis developing caused by the overproduction of bacterial Helicobacter pylori (H. pylori) in induced infection, have been inclusive.
Coffee, a popular and social beverage all over the world, particularly in the West, is a drink made from roasted beans from the Coffea plant, native to tropical Africa and Madagascar.
Gastritis is a condition of inflammation, irritation, or erosion of the stomach lining caused by bacterial Helicobacter pylori (H. pylori) overexpression located at the lining of the stomach.
In a study of a total of 447 patients aged 15-79 had not had peptic ulcer disease or treatment for H pylori infection. researchers found that coffee consumption showed a positive dose-response relation in the reduced active stomach lining infection and after adjusted odds ratios, patients who drank < 3 cups and > or = 3 cups of coffee per day displayed the relative risk ratio of 1.49 and 2.49 respectively, in compared to non-drinkers.
The result suggested that coffee intake showed negative effects in attenuated in the course of H pylori-induced active infection. In other words, coffee intake may elevate active infection caused by bacterial H pylori.
Another, in the study of a sample of 238 subjects (hospital employees) initially (in 1994) seronegative to H. pylori tested again 5 years later (1999), in two different periods (1994 and 1999). returned questionnaire from patients showed a neutral effect between coffee consumption and risk of infection caused by H. pylori regardless of the number of cups intake daily and after taking into account of other factors.
More importantly, in the investigation of the effect of coffee consumption on four major acid-related diseases: gastric ulcer (GU), a serve case of gastric infection, duodenal ulcer (DU), reflux esophagitis (RE), and non-erosive reflux disease (NERD) based on the large-scale multivariate analysis of the 9,517 healthy adults, researchers filed the following results in related to coffee intake and risk of Helicobacter pylori (HP) infection
1. Coffee consumption expressed a relatively low ratio of pepsinogen I/II
2. Coffee intake expressed a positively related risk of Helicobacter pylori (HP) infection by multiple logistic regression analysis,
3. Coffee intake was not associated with either GU or DU
But after taking into account other risk factors, researchers exemplified that " there is no significant relationship between coffee consumption and the four major acid-related upper gastrointestinal disorders".
With all the information collected, coffee intake may have a very significant and neutral link to an increased risk of Gastritis caused by bacterial infection, therefore, clarification by large sample size and cohost studies are necessary,
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Author Biography
Kyle J. Norton, Master of Nutrients
Health article writer and researcher; Over 10.000 articles and research papers have been written and published online, including worldwide health, ezine articles, article base, health blogs, self-growth, best before it's news, the Karate GB Daily, etc.,.
Named TOP 50 MEDICAL ESSAYS FOR ARTISTS & AUTHORS TO READ by Disilgold.com Named 50 of the best health Tweeters Canada - Huffington Post
Nominated for shorty award over last 4 years
Some articles have been used as references in medical research, such as the international journal Pharma and Bio Science, ISSN 0975-6299.
Sources
(1) Relation of smoking and alcohol and coffee consumption to active Helicobacter pylori infection: cross sectional study by Brenner H1, Rothenbacher D, Bode G, Adler G.(PubMed)
(2) Relationship of smoking and coffee and alcohol consumption with seroconversion to Helicobacter pylori: a longitudinal study in hospital workers by Gikas A1, Triantafillidis JK, Apostolidis N, Mallas E, Peros G, Androulakis G.(PubMed)
(3) No association of coffee consumption with gastric ulcer, duodenal ulcer, reflux esophagitis, and non-erosive reflux disease: a cross-sectional study of 8,013 healthy subjects in Japan by Shimamoto T1, Yamamichi N, Kodashima S, Takahashi Y, Fujishiro M, Oka M, Mitsushima T, Koike K(PubMed)
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